Area of research
Surgery · Immunology
Research interest
Research interests include Lipoproteins and Cardiovascular Health, Atherosclerosis and Cardiovascular Diseases, Cancer, Lipids, and Metabolism, and Diabetes, Cardiovascular Risks, and Lipoproteins.
<b>AHA PREVENT Equations and Lipoprotein(a) for Cardiovascular Disease Risk </b>
The effects of colchicine on lipoprotein(a)- and oxidized phospholipid-associated cardiovascular disease risk
Lipoprotein(a) Testing Trends in a Large Academic Health System in the United States
Lipoprotein(a) and carotid intima-media thickness in children with familial hypercholesterolaemia in the Netherlands: a 20-year follow-up study
Lipoprotein(a) and the pooled cohort equations for ASCVD risk prediction: The Multi-Ethnic Study of Atherosclerosis
Effect of Pelacarsen on Lipoprotein(a) Cholesterol and Corrected Low-Density Lipoprotein Cholesterol
DNA methyltransferase 3 alpha and TET methylcytosine dioxygenase 2 restrain mitochondrial DNA-mediated interferon signaling in macrophages
Novel method for quantification of lipoprotein(a)-cholesterol: implications for improving accuracy of LDL-C measurements
Clonal hematopoiesis driven by DNMT3A and TET2 mutations: role in monocyte and macrophage biology and atherosclerotic cardiovascular disease
Atherogenic Lipoprotein(a) Increases Vascular Glycolysis, Thereby Facilitating Inflammation and Leukocyte Extravasation
Low‐Density Lipoprotein Cholesterol Corrected for Lipoprotein(a) Cholesterol, Risk Thresholds, and Cardiovascular Events
ApoCIII-Lp(a) complexes in conjunction with Lp(a)-OxPL predict rapid progression of aortic stenosis
Statin therapy increases lipoprotein(a) levels
Potent reduction of plasma lipoprotein (a) with an antisense oligonucleotide in human subjects does not affect ex vivo fibrinolysis
Oxidized phospholipids are proinflammatory and proatherogenic in hypercholesterolaemic mice
Association of Mild to Moderate Aortic Valve Stenosis Progression With Higher Lipoprotein(a) and Oxidized Phospholipid Levels
Reduction of myocardial ischaemia–reperfusion injury by inactivating oxidized phospholipids
Relationship between “LDL-C”, estimated true LDL-C, apolipoprotein B-100, and PCSK9 levels following lipoprotein(a) lowering with an antisense oligonucleotide
Lipoprotein(a)-Associated Molecules Are Prominent Components in Plasma and Valve Leaflets in Calcific Aortic Valve Stenosis
Oxidized Phospholipids on Lipoprotein(a) Elicit Arterial Wall Inflammation and an Inflammatory Monocyte Response in Humans
Effect of therapeutic interventions on oxidized phospholipids on apolipoprotein B100 and lipoprotein(a)
PCSK9 Association With Lipoprotein(a)
Lipoprotein(a) and oxidized phospholipids in calcific aortic valve stenosis
Oxidized Phospholipids, Lipoprotein(a), and Progression of Calcific Aortic Valve Stenosis
‘LDL-C’ = LDL-C + Lp(a)-C