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Ralph Tiedt

Yale Cancer Center · US
Area of research
Pulmonary and Respiratory Medicine · Molecular Biology
Research interest
Research focused on Nausea and Cancer research, with related work in Fibroblast growth factor receptor, Triple-negative breast cancer, Insertional mutagenesis. Notable publications include 'Polyclonal Secondary FGFR2 Mutations Drive Acquired Resistance to FGFR Inhibition in Patients with FGFR2 Fusion–Positive Cholangiocarcinoma', 'Increased lysosomal biomass is responsible for the resistance of triple-negative breast cancers to CDK4/6 inhibition', and 'Resistance mechanisms to TP53-MDM2 inhibition identified by in vivo piggyBac transposon mutagenesis screen in an Arf −/− mouse model'.
h-index
citations
826
works
7
NIH funding
primary concept
email

Recent publications

Increased lysosomal biomass is responsible for the resistance of triple-negative breast cancers to CDK4/6 inhibition
Science Advances 2020cited by 105position: middledoi
A phase II study of the efficacy and safety of the MET inhibitor capmatinib (INC280) in patients with advanced hepatocellular carcinoma
Therapeutic Advances in Medical Oncology 2019cited by 58position: middledoi
A Phase Ib/II, open-label, multicenter study of INC280 (capmatinib) alone and in combination with buparlisib (BKM120) in adult patients with recurrent glioblastoma
Journal of Neuro-Oncology 2019cited by 49position: middledoi
A conditional inducible JAK2V617F transgenic mouse model reveals myeloproliferative disease that is reversible upon switching off transgene expression
PLoS ONE 2019cited by 11position: middledoi
Resistance mechanisms to TP53-MDM2 inhibition identified by in vivo piggyBac transposon mutagenesis screen in an Arf <sup>−/−</sup> mouse model
Proceedings of the National Academy of Sciences 2017cited by 62position: middledoi
ACTR-74. A PHASE IB/II, OPEN-LABEL, MULTICENTER STUDY OF CAPMATINIB (INC280) ALONE AND IN COMBINATION WITH BUPARLISIB (BKM120) IN ADULT PATIENTS WITH RECURRENT GLIOBLASTOMA
Neuro-Oncology 2017cited by 1position: middledoi
Polyclonal Secondary <i>FGFR2</i> Mutations Drive Acquired Resistance to FGFR Inhibition in Patients with FGFR2 Fusion–Positive Cholangiocarcinoma
Cancer Discovery 2016cited by 540position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

Ghazaleh Tabatabai · Goethe University Frankfurt2 papers (2017–2019)Sylvia Zhao · Novartis (China)2 papers (2017–2019)Francesco Hofmann · Novartis (Switzerland)2 papers (2017–2019)W.K. Alfred Yung · The University of Texas MD Anderson Cancer Center2 papers (2017–2019)Analía Azaro · Novartis (Switzerland)2 papers (2017–2019)Sergio Vicente · Novartis (Switzerland)2 papers (2017–2019)Martin J. van den Bent · St James's University Hospital2 papers (2017–2019)Patrick Y. Wen · Harvard University2 papers (2017–2019)Markus Joerger · Palmetto Hematology Oncology2 papers (2017–2019)Filip De Vos · Leidsche Rijn Julius Health Centers2 papers (2017–2019)Jordi Rodón · University of California San Francisco Medical Center2 papers (2017–2019)Tiina Kirsilae · Novartis (Switzerland)2 papers (2017–2019)Emeline Mandon · Novartis (Switzerland)2 papers (2017–2019)Andrew B. Lassman · Columbia University2 papers (2017–2019)Vincent Romanet · Novartis (Switzerland)2 papers (2017–2019)Juan Manuel Sepúlveda-Sánchez · Research Institute Hospital 12 de Octubre2 papers (2017–2019)Wolfgang Wick · Heidelberger Institut für Radioonkologie2 papers (2017–2019)Masato Murakami · Novartis (Switzerland)2 papers (2017–2019)Emilie A. Chapeau · Novartis (Switzerland)2 papers (2017–2019)Antoine de Weck · Centre for Human Genetics1 papers (2017–2017)