Area of research
Hematology · Public Health, Environmental and Occupational Health
Research interest
Research interests include Acute Myeloid Leukemia Research, Acute Lymphoblastic Leukemia research, Retinoids in leukemia and cellular processes, and Cancer-related molecular mechanisms research.
Epidemiology, clinical features, and molecular basis of <i>TTMV</i> :: <i>RARA</i> -driven acute promyelocytic leukemia
Cystine-Modified Lignin–Copper Coordination Nanocarriers Improve the Therapeutic Efficacy of Tyrosine Kinase Inhibition via Cuproptosis
The genomic landscape of sensitivity to arsenic trioxide uncovered by genome-wide CRISPR-Cas9 screening
A multicenter phase <scp>II</scp> trial of primary prophylactic <scp>PEG‐rhG‐CSF</scp> in pediatric patients with solid tumors and <scp>non‐Hodgkin</scp> lymphoma after chemotherapy: An interim analysis
Chromatin-associated orphan snoRNA regulates DNA damage-mediated differentiation via a non-canonical complex
Co-expression of a PD-L1-specific chimeric switch receptor augments the efficacy and persistence of CAR T cells via the CD70-CD27 axis
Disruption of CISH promotes the antitumor activity of human T cells and decreases PD-1 expression levels
The snoRNA-like lncRNA LNC-SNO49AB drives leukemia by activating the RNA-editing enzyme ADAR1
A Nomogram for Predicting Event-Free Survival in Childhood Acute Lymphoblastic Leukemia: A Multicenter Retrospective Study
The oncomicropeptide APPLE promotes hematopoietic malignancy by enhancing translation initiation
Functional Characteristics and Application of Mesenchymal Stem Cells in Systemic Lupus Erythematosus
Prognostic Value and Outcome for ETV6/RUNX1-Positive Pediatric Acute Lymphoblastic Leukemia: A Report From the South China Children’s Leukemia Group
Performance of PRISM III, PELOD-2, and P-MODS Scores in Two Pediatric Intensive Care Units in China
Abnormal thymic B cell activation and impaired T cell differentiation in pristane-induced lupus mice
The lncRNA LAMP5-AS1 drives leukemia cell stemness by directly modulating DOT1L methyltransferase activity in MLL leukemia
Arsenic trioxide and all-trans retinoic acid suppress the expression of FLT3-ITD
circMYBL2, a circRNA from MYBL2, regulates FLT3 translation by recruiting PTBP1 to promote FLT3-ITD AML progression
circRNA circAF4 functions as an oncogene to regulate MLL-AF4 fusion protein expression and inhibit MLL leukemia progression
LncRNA ANRIL regulates AML development through modulating the glucose metabolism pathway of AdipoR1/AMPK/SIRT1
MIR-708 promotes phagocytosis to eradicate T-ALL cells by targeting CD47
The lncRNA HOTAIRM1 regulates the degradation of PML-RARA oncoprotein and myeloid cell differentiation by enhancing the autophagy pathway