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Sebastian Markmiller

University of California, San Diego · US
Area of research
Molecular Biology · Neurology
Research interest
Research interests include RNA Research and Splicing, Amyotrophic Lateral Sclerosis Research, RNA modifications and cancer, and Neurogenetic and Muscular Disorders Research.
h-index
16
citations
3,349
works
30
NIH funding
primary concept
email

Recent publications

Huntington’s disease mice and human brain tissue exhibit increased G3BP1 granules and TDP43 mislocalization
Journal of Clinical Investigation 2021cited by 66position: middledoi
Persistent mRNA localization defects and cell death in ALS neurons caused by transient cellular stress
Cell Reports 2021cited by 39position: firstdoi
Large-scale tethered function assays identify factors that regulate mRNA stability and translation
Nature Structural & Molecular Biology 2020cited by 133position: middledoi
Small-Molecule Modulation of TDP-43 Recruitment to Stress Granules Prevents Persistent TDP-43 Accumulation in ALS/FTD
Neuron 2019cited by 270position: middledoi
Phenotypic Suppression of ALS/FTD-Associated Neurodegeneration Highlights Mechanisms of Dysfunction
Journal of Neuroscience 2019cited by 16position: middledoi
Context-Dependent and Disease-Specific Diversity in Protein Interactions within Stress Granules
Cell 2018cited by 951position: firstdoi
Elimination of Toxic Microsatellite Repeat Expansion RNA by RNA-Targeting Cas9
Cell 2017cited by 256position: middledoi
Biallelic mutations in the 3′ exonuclease TOE1 cause pontocerebellar hypoplasia and uncover a role in snRNA processing
Nature Genetics 2017cited by 101position: middledoi
Programmable RNA Tracking in Live Cells with CRISPR/Cas9
Cell 2016cited by 533position: middledoi
Minor class splicing shapes the zebrafish transcriptome during development
Proceedings of the National Academy of Sciences 2014cited by 72position: firstdoi
Targeted degradation of sense and antisense <i>C9orf72</i> RNA foci as therapy for ALS and frontotemporal degeneration
Proceedings of the National Academy of Sciences 2013cited by 554position: middledoi
Autophagy Induction Is a Tor- and Tp53-Independent Cell Survival Response in a Zebrafish Model of Disrupted Ribosome Biogenesis
PLoS Genetics 2013cited by 79position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

G Yeo · Aarhus University Hospital7 papers (2016–2021)Mark Y. Fang · BGI Group (China)3 papers (2016–2019)Éric Lécuyer · Centre National de la Recherche Scientifique3 papers (2018–2021)Kari L. Server · University of California San Diego2 papers (2018–2021)Heather Verkade · The University of Melbourne2 papers (2013–2014)Eric J. Bennett · University of California San Diego2 papers (2018–2021) · 2 papers (2013–2014)Didier Y. R. Stainier · Max Planck Institute for Heart and Lung Research2 papers (2013–2014)Ashkan Javaherian · Gladstone Institutes2 papers (2019–2021) · 2 papers (2013–2014)Anthony Q. Vu · University of California San Diego2 papers (2019–2021) · 2 papers (2013–2014)David A. Nelles · La Jolla Bioengineering Institute2 papers (2016–2017)Elke A. Ober · Friedrich-Alexander-Universität Erlangen-Nürnberg2 papers (2013–2014)Graham J. Lieschke · Monash University2 papers (2013–2014)Shashank Sathe · Sanford Consortium for Regenerative Medicine2 papers (2020–2021)Steven Finkbeiner · Gladstone Institutes2 papers (2019–2021)Holly A. Field · University of California, San Francisco2 papers (2013–2014)Thai B. Nguyen · University of California, Irvine2 papers (2021–2021) · 2 papers (2013–2014)