Area of research
Immunology · Molecular Biology
Research interest
Research focused on T-cell receptor and Major histocompatibility complex, with related work in Virology, Antigen, Epitope. Notable publications include 'T Cell Antigen Receptor Recognition of Antigen-Presenting Molecules', 'Immune self-reactivity triggered by drug-modified HLA-peptide repertoire', and 'Human CD8+ T cell cross-reactivity across influenza A, B and C viruses'.
Type I interferons induce an epigenetically distinct memory B cell subset in chronic viral infection
A common allele of HLA is associated with asymptomatic SARS-CoV-2 infection
Canonical T cell receptor docking on peptide–MHC is essential for T cell signaling
A Natural Peptide Antigen within the Plasmodium Ribosomal Protein RPL6 Confers Liver TRM Cell-Mediated Immunity against Malaria in Mice
Human CD8+ T cell cross-reactivity across influenza A, B and C viruses
T cell autoreactivity directed toward CD1c itself rather than toward carried self lipids
A T-cell receptor escape channel allows broad T-cell response to CD1b and membrane phospholipids
A molecular basis of human T cell receptor autoreactivity toward self-phospholipids
T cell receptor recognition of CD1b presenting a mycobacterial glycolipid
Targeted suppression of autoreactive CD8+ T-cell activation using blocking anti-CD8 antibodies
HIV-1–Specific CD8 T Cells Exhibit Limited Cross-Reactivity during Acute Infection
T Cell Antigen Receptor Recognition of Antigen-Presenting Molecules
Preexisting CD8 <sup>+</sup> T-cell immunity to the H7N9 influenza A virus varies across ethnicities
TCR Bias and Affinity Define Two Compartments of the CD1b–Glycolipid-Specific T Cell Repertoire
CD1a-autoreactive T cells recognize natural skin oils that function as headless antigens
Cutting Edge: CD1a Tetramers and Dextramers Identify Human Lipopeptide–Specific T Cells Ex Vivo
Immune self-reactivity triggered by drug-modified HLA-peptide repertoire
Structural basis for the killing of human beta cells by CD8+ T cells in type 1 diabetes